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dc.contributor.authorOlsson, AY
dc.contributor.authorCooper, CS
dc.date.accessioned2018-09-05T15:09:28Z
dc.date.issued2005-11
dc.identifier11
dc.identifier.citationBRITISH JOURNAL OF HOSPITAL MEDICINE, 2005, 66 pp. 612 - 616
dc.identifier.issn1462-3935
dc.identifier.urihttps://repository.icr.ac.uk/handle/internal/2564
dc.identifier.doi10.12968/hmed.2005.66.11.20021
dc.description.abstractCancer involves accumulation of genetic alterations. This review highlights the alterations in control pathways for cell division, development, DNA repair, angiogenesis and cell death that are believed to be key players in the development of prostate cancer.
dc.format.extent612 - 616
dc.languageeng
dc.language.isoeng
dc.publisherMA HEALTHCARE LTD
dc.titleThe molecular basis of prostate cancer
dc.typeJournal Article
rioxxterms.versionofrecord10.12968/hmed.2005.66.11.20021
rioxxterms.licenseref.startdate2005-11
rioxxterms.typeJournal Article/Review
dc.relation.isPartOfBRITISH JOURNAL OF HOSPITAL MEDICINE
pubs.notesaffiliation: Male Urol Canc Res Ctr, Inst Canc Res, Sutton SM2 5NG, Surrey, England. keywords-plus: ANTIANDROGEN THERAPY; RESISTANCE; PROLIFERATION; PROGRESSION; EXPRESSION; APOPTOSIS; PATHWAY; CELLS research-areas: General & Internal Medicine web-of-science-categories: Medicine, General & Internal number-of-cited-references: 19 times-cited: 3 usage-count-last-180-days: 0 usage-count-since-2013: 0 journal-iso: Br. J. Hosp. Med. doc-delivery-number: 019BG unique-id: ISI:000235810000003 da: 2018-09-04
pubs.notesNot known
pubs.organisational-group/ICR
pubs.organisational-group/ICR/Primary Group
pubs.organisational-group/ICR/Primary Group/ICR Divisions
pubs.organisational-group/ICR/Primary Group/ICR Divisions/Closed research teams
pubs.organisational-group/ICR/Primary Group/ICR Divisions/Closed research teams/Cell Transformation
pubs.organisational-group/ICR
pubs.organisational-group/ICR/Primary Group
pubs.organisational-group/ICR/Primary Group/ICR Divisions
pubs.organisational-group/ICR/Primary Group/ICR Divisions/Closed research teams
pubs.organisational-group/ICR/Primary Group/ICR Divisions/Closed research teams/Cell Transformation
pubs.volume66
pubs.embargo.termsNot known
icr.researchteamCell Transformationen_US
dc.contributor.icrauthorCooper, Colinen


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