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dc.contributor.authorPeto, Jen_US
dc.date.accessioned2018-09-17T15:18:25Z
dc.date.issued2002-06en_US
dc.identifier5en_US
dc.identifier.citationCANCER CELL, 2002, 1 pp. 411 - 412en_US
dc.identifier.issn1535-6108en_US
dc.identifier.urihttps://repository.icr.ac.uk/handle/internal/2751
dc.identifier.doi10.1016/S1535-6108(02)00079-Xen_US
dc.description.abstractA polygenic model in which many individually weak genes combine multiplicately to cause a 50-fold range of risk in the population explains several puzzling aspects of familial breast cancer epidemiology, including the very high risk in some families and the failure to identify important new genes since the discovery of BRCA1 and BRCA2.en_US
dc.format.extent411 - 412en_US
dc.titleBreast cancer susceptibility - A new look at an old modelen_US
dc.typeJournal Article
rioxxterms.versionofrecord10.1016/S1535-6108(02)00079-Xen_US
rioxxterms.licenseref.startdate2002-06en_US
rioxxterms.typeJournal Article/Reviewen_US
dc.relation.isPartOfCANCER CELLen_US
pubs.notesunique-id: ISI:000178352300004en_US
pubs.notesNot knownen_US
pubs.organisational-group/ICR
pubs.volume1en_US
pubs.embargo.termsNot knownen_US
dc.contributor.icrauthorPeto, Julianen_US


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