Cell Competition in Carcinogenesis.

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Embargo End Date

ICR Authors

Authors

Madan, E
Palma, AM
Vudatha, V
Trevino, JG
Natarajan, KN
Winn, RA
Won, KJ
Graham, TA
Drapkin, R
McDonald, SAC
Fisher, PB
Gogna, R

Document Type

Journal Article

Date

2022-12-16

Date Accepted

2022-09-29

Abstract

The majority of human cancers evolve over time through the stepwise accumulation of somatic mutations followed by clonal selection akin to Darwinian evolution. However, the in-depth mechanisms that govern clonal dynamics and selection remain elusive, particularly during the earliest stages of tissue transformation. Cell competition (CC), often referred to as 'survival of the fittest' at the cellular level, results in the elimination of less fit cells by their more fit neighbors supporting optimal organism health and function. Alternatively, CC may allow an uncontrolled expansion of super-fit cancer cells to outcompete their less fit neighbors thereby fueling tumorigenesis. Recent research discussed herein highlights the various non-cell-autonomous principles, including interclonal competition and cancer microenvironment competition supporting the ability of a tumor to progress from the initial stages to tissue colonization. In addition, we extend current insights from CC-mediated clonal interactions and selection in normal tissues to better comprehend those factors that contribute to cancer development.

Citation

Cancer Research, 2022, pp. CAN-22-2217 -

Source Title

Cancer Research

Publisher

AMER ASSOC CANCER RESEARCH

ISSN

0008-5472

eISSN

1538-7445
1538-7445

Research Team

Genomics & evolut dynam

Notes