Deregulation of the Egfr/Ras Signaling Pathway Induces Age-related Brain Degeneration in the<i>Drosophila</i>Mutant<i>vap</i>

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Authors

Botella, JA
Kretzschmar, D
Kiermayer, C
Feldmann, P
Hughes, DA
Schneuwly, S

Document Type

Journal Article

Date

2003-01-01

Date Accepted

Abstract

<jats:p>Ras signaling has been shown to play an important role in promoting cell survival in many different tissues. Here we show that upregulation of Ras activity in adult Drosophila neurons induces neuronal cell death, as evident from the phenotype of vacuolar peduncle (vap) mutants defective in theDrosophila RasGAP gene, which encodes a Ras GTPase-activating protein. These mutants show age-related brain degeneration that is dependent on activation of the EGF receptor signaling pathway in adult neurons, leading to autophagic cell death (cell death type 2). These results provide the first evidence for a requirement of Egf receptor activity in differentiated adultDrosophila neurons and show that a delicate balance of Ras activity is essential for the survival of adult neurons.</jats:p>

Citation

MOLECULAR BIOLOGY OF THE CELL, 2003, 14 pp. 241 - 250

Source Title

Publisher

American Society for Cell Biology (ASCB)

ISSN

1059-1524

eISSN

Research Team

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