Now showing items 1-2 of 2

    • Lack of association between modifiable exposures and glioma risk: a Mendelian randomization analysis. 

      Saunders, CN; Cornish, AJ; Kinnersley, B; Law, PJ; Claus, EB; Il'yasova, D; Schildkraut, J; Barnholtz-Sloan, JS; Olson, SH; Bernstein, JL; Lai, RK; Chanock, S; Rajaraman, P; Johansen, C; Jenkins, RB; Melin, BS; Wrensch, MR; Sanson, M; Bondy, ML; Houlston, RS (2020-02)
      <h4>Background</h4>The etiological basis of glioma is poorly understood. We have used genetic markers in a Mendelian randomization (MR) framework to examine if lifestyle, cardiometabolic, and inflammatory factors influence ...
    • Searching for causal relationships of glioma: a phenome-wide Mendelian randomisation study. 

      Saunders, CN; Cornish, AJ; Kinnersley, B; Law, PJ; Houlston, RS; Collaborators (2021-01)
      <h4>Background</h4>The aetiology of glioma is poorly understood. Summary data from genome-wide association studies (GWAS) can be used in a Mendelian randomisation (MR) phenome-wide association study (PheWAS) to search for ...