| dc.contributor.author | Conway, O | |
| dc.contributor.author | Kirkin, V | |
| dc.date.accessioned | 2017-07-20T10:07:01Z | |
| dc.date.issued | 2017-05-01 | |
| dc.identifier.citation | Cell research, 2017, 27 (5), pp. 595 - 597 | |
| dc.identifier.issn | 1001-0602 | |
| dc.identifier.uri | https://repository.icr.ac.uk/handle/internal/733 | |
| dc.identifier.eissn | 1748-7838 | |
| dc.identifier.doi | 10.1038/cr.2017.56 | |
| dc.description.abstract | The multimodular adapter p62/sequestosome-1 plays prominent roles in physiology and disease by mediating cell signaling and cargo degradation. The work by Peng et al. published recently in Cell Research provides mechanistic insights into activation of its autophagy receptor function critical for maintaining cell homeostasis during various forms of stress. | |
| dc.format | Print-Electronic | |
| dc.format.extent | 595 - 597 | |
| dc.language | eng | |
| dc.language.iso | eng | |
| dc.publisher | INST BIOCHEMISTRY & CELL BIOLOGY | |
| dc.rights.uri | https://www.rioxx.net/licenses/all-rights-reserved | |
| dc.title | Love laughs at Locksmiths: Ubiquitylation of p62 unlocks its autophagy receptor potential. | |
| dc.type | Journal Article | |
| dcterms.dateAccepted | 2017-05-01 | |
| rioxxterms.versionofrecord | 10.1038/cr.2017.56 | |
| rioxxterms.licenseref.uri | https://www.rioxx.net/licenses/all-rights-reserved | |
| rioxxterms.licenseref.startdate | 2017-05 | |
| rioxxterms.type | Journal Article/Review | |
| dc.relation.isPartOf | Cell research | |
| pubs.issue | 5 | |
| pubs.notes | Not known | |
| pubs.organisational-group | /ICR | |
| pubs.organisational-group | /ICR/Primary Group | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Breast Cancer Research | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Breast Cancer Research/Cell Death and Immunity | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics/Cancer Pharmacology & Stress Response (CPSR) | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics/Cancer Pharmacology & Stress Response (CPSR) | |
| pubs.organisational-group | /ICR | |
| pubs.organisational-group | /ICR/Primary Group | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Breast Cancer Research | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Breast Cancer Research/Cell Death and Immunity | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics/Cancer Pharmacology & Stress Response (CPSR) | |
| pubs.organisational-group | /ICR/Primary Group/ICR Divisions/Cancer Therapeutics/Cancer Pharmacology & Stress Response (CPSR) | |
| pubs.publication-status | Published | |
| pubs.volume | 27 | |
| pubs.embargo.terms | Not known | |
| icr.researchteam | Cell Death and Immunity | |
| icr.researchteam | Cancer Pharmacology & Stress Response (CPSR) | |
| dc.contributor.icrauthor | Conway, Owen | |
| dc.contributor.icrauthor | Kirkin, Vladimir | |
